Title : Endoplasmic Reticulum Stress in Subepithelial Myofibroblasts Increases the TGF-β1 Activity That Regulates Fibrosis in Crohn's Disease.

Pub. Date : 2020 May 12

PMID : 32031621






1 Functional Relationships(s)
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1 Tunicamycin elicited time-dependent increase in GRP78 protein levels, direct interaction with latent TGF-beta1, and activated TGF-beta1 signaling. Tunicamycin heat shock protein family A (Hsp70) member 5 Homo sapiens