Title : Calpain-1 ablation partially rescues disease-associated hallmarks in models of Machado-Joseph disease.

Pub. Date : 2020 Apr 15

PMID : 31960910






1 Functional Relationships(s)
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1 Proteolytic fragmentation of polyglutamine-expanded ataxin-3 is a concomitant and modifier of the molecular pathogenesis of Machado-Joseph disease (MJD), the most common autosomal dominant cerebellar ataxia. polyglutamine ataxin 3 Mus musculus