Title : High glucose concentrations mask cellular phenotypes in a stem cell model of tuberous sclerosis complex.

Pub. Date : 2019 Dec

PMID : 31761686






1 Functional Relationships(s)
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1 As TSC is potentially caused by a disruption of the mechanistic target of rapamycin (mTOR) pathway, a main integrator of metabolic information and intracellular signaling, we aimed to examine the impact of different glucose concentrations in the culture media on cellular phenotypes implicated in tuber characteristics. Glucose TSC complex subunit 1 Homo sapiens