Title : The Mechanisms Underlying the Cytotoxic Effects of Copper Via Differentiated Embryonic Chondrocyte Gene 1.

Pub. Date : 2019 Oct 22

PMID : 31652494






1 Functional Relationships(s)
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1 Copper sulfate also increased the levels of apoptosis, senescence, mitochondrial dysfunction, autophagy, ROS, and the expression of several stress proteins, including ATF3, c-Fos, DEC1 (differentiated embryonic chondrocyte gene 1), p21, p53, and HIF-1alpha (hypoxia-inducible factor 1 alpha). Copper Sulfate H3 histone pseudogene 16 Homo sapiens