Title : Boiled Abalone Byproduct Peptide Exhibits Anti-Tumor Activity in HT1080 Cells and HUVECs by Suppressing the Metastasis and Angiogenesis in Vitro.

Pub. Date : 2019 Aug 14

PMID : 31343893






4 Functional Relationships(s)
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Sentence
Compound Name
Protein Name
Organism
1 BABP inhibits phorbol 12-myristate 13-acetate (PMA)-induced matrix metalloproteinase (MMP) expression and activity by blocking mitogen-activated protein kinases (MAPKs) and NF-kappaB signaling and hypoxia-induced vascular endothelial growth factor (VEGF) secretion and hypoxia inducible factor (HIF)-1alpha accumulation through suppressing the AKT/mTOR signal pathway. Tetradecanoylphorbol Acetate vascular endothelial growth factor A Homo sapiens
2 BABP inhibits phorbol 12-myristate 13-acetate (PMA)-induced matrix metalloproteinase (MMP) expression and activity by blocking mitogen-activated protein kinases (MAPKs) and NF-kappaB signaling and hypoxia-induced vascular endothelial growth factor (VEGF) secretion and hypoxia inducible factor (HIF)-1alpha accumulation through suppressing the AKT/mTOR signal pathway. Tetradecanoylphorbol Acetate vascular endothelial growth factor A Homo sapiens
3 BABP inhibits phorbol 12-myristate 13-acetate (PMA)-induced matrix metalloproteinase (MMP) expression and activity by blocking mitogen-activated protein kinases (MAPKs) and NF-kappaB signaling and hypoxia-induced vascular endothelial growth factor (VEGF) secretion and hypoxia inducible factor (HIF)-1alpha accumulation through suppressing the AKT/mTOR signal pathway. Tetradecanoylphorbol Acetate vascular endothelial growth factor A Homo sapiens
4 BABP inhibits phorbol 12-myristate 13-acetate (PMA)-induced matrix metalloproteinase (MMP) expression and activity by blocking mitogen-activated protein kinases (MAPKs) and NF-kappaB signaling and hypoxia-induced vascular endothelial growth factor (VEGF) secretion and hypoxia inducible factor (HIF)-1alpha accumulation through suppressing the AKT/mTOR signal pathway. Tetradecanoylphorbol Acetate vascular endothelial growth factor A Homo sapiens