Title : Neuroinflammation induced by lipopolysaccharide causes cognitive impairment in mice.

Pub. Date : 2019 Apr 8

PMID : 30962497






2 Functional Relationships(s)
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1 The levels of interleukin-4 (IL-4) and interleukin-10 (IL-10) in the serum and brain homogenates were reduced by the LPS treatment, while the levels of tumor necrosis factor-alpha (TNF-alpha), interleukin-1beta (IL-1beta), prostaglandin E2 (PGE2) and nitric oxide (NO) were increased. Dinoprostone tumor necrosis factor Mus musculus
2 The levels of interleukin-4 (IL-4) and interleukin-10 (IL-10) in the serum and brain homogenates were reduced by the LPS treatment, while the levels of tumor necrosis factor-alpha (TNF-alpha), interleukin-1beta (IL-1beta), prostaglandin E2 (PGE2) and nitric oxide (NO) were increased. Dinoprostone tumor necrosis factor Mus musculus