Title : Isofraxidin attenuates IL-1β-induced inflammatory response in human nucleus pulposus cells.

Pub. Date : 2019 Aug

PMID : 30891836






2 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 The results proved that isofraxidin attenuated the IL-1beta-induced significant increases in inflammatory mediators and cytokines including nitric oxide (NO), inducible NO synthase (iNOS), cyclooxygenase-2 (COX-2), prostaglandin E2 (PGE2), tumor necrosis factor alpha (TNF-alpha), and IL-6. Dinoprostone interleukin 1 beta Homo sapiens
2 The results proved that isofraxidin attenuated the IL-1beta-induced significant increases in inflammatory mediators and cytokines including nitric oxide (NO), inducible NO synthase (iNOS), cyclooxygenase-2 (COX-2), prostaglandin E2 (PGE2), tumor necrosis factor alpha (TNF-alpha), and IL-6. Dinoprostone interleukin 1 beta Homo sapiens