Title : Glycyrrhetinic Acid Improves Insulin-Response Pathway by Regulating the Balance between the Ras/MAPK and PI3K/Akt Pathways.

Pub. Date : 2019 Mar 12

PMID : 30871060






3 Functional Relationships(s)
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1 GA also inhibited the PMA-induced phosphorylation of IkappaB kinase alpha/beta (IKKalpha/beta), c-Jun N-terminal kinase (JNK) and p38 proteins (P38), suggesting that IKKalpha/beta, JNK and P38 activation is dependent on PKC activity. Tetradecanoylphorbol Acetate mitogen-activated protein kinase 8 Homo sapiens
2 GA also inhibited the PMA-induced phosphorylation of IkappaB kinase alpha/beta (IKKalpha/beta), c-Jun N-terminal kinase (JNK) and p38 proteins (P38), suggesting that IKKalpha/beta, JNK and P38 activation is dependent on PKC activity. Tetradecanoylphorbol Acetate mitogen-activated protein kinase 8 Homo sapiens
3 GA also inhibited the PMA-induced phosphorylation of IkappaB kinase alpha/beta (IKKalpha/beta), c-Jun N-terminal kinase (JNK) and p38 proteins (P38), suggesting that IKKalpha/beta, JNK and P38 activation is dependent on PKC activity. Tetradecanoylphorbol Acetate mitogen-activated protein kinase 8 Homo sapiens