Title : Curcumin attenuates palmitic acid-induced cell apoptosis by inhibiting endoplasmic reticulum stress in H9C2 cardiomyocytes.

Pub. Date : 2019 Jun

PMID : 30859861






4 Functional Relationships(s)
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1 Treatment with PA reduced cell viability, induced apoptosis, enhanced apoptosis-related protein expression (Caspase 3 and BCL-2 associated X protein (BAX)), and activated ER stress marker protein expression (glucose-regulated protein 78 (GRP78) and CCAAT/enhancer binding protein homologous protein (CHOP)). Palmitic Acid DNA-damage inducible transcript 3 Rattus norvegicus
2 Treatment with PA reduced cell viability, induced apoptosis, enhanced apoptosis-related protein expression (Caspase 3 and BCL-2 associated X protein (BAX)), and activated ER stress marker protein expression (glucose-regulated protein 78 (GRP78) and CCAAT/enhancer binding protein homologous protein (CHOP)). Palmitic Acid DNA-damage inducible transcript 3 Rattus norvegicus
3 Curcumin attenuated PA-induced reduction in cell viability and activation of apoptosis, Caspase 3 activity, BAX, CHOP, and GRP78 expression. Palmitic Acid DNA-damage inducible transcript 3 Rattus norvegicus
4 Both curcumin and 4-PBA also attenuated PA-induced increase in ER stress protein (CHOP and GRP78) expression. Palmitic Acid DNA-damage inducible transcript 3 Rattus norvegicus