Title : Post-translational processing and activation of insulin and EGF proreceptors.

Pub. Date : 1985 Oct-Nov

PMID : 3000457






1 Functional Relationships(s)
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1 Thus, treatment of 3T3-L1 adipocytes with tunicamycin caused the loss of cellular insulin binding activity and the accumulation of an inactive aglyco-proreceptor. Tunicamycin insulin Homo sapiens