Title : Stiff person case misdiagnosed as conversion disorder: A case report.

Pub. Date : 2017 Fall

PMID : 29201327






6 Functional Relationships(s)
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Protein Name
Organism
1 The pathophysiology of the disease is caused by the decreased level of the glutamic acid decarboxylase (GAD) activity due to an autoantibody against GAD that decreases the level of gamma-aminobutyric acid (GABA). gamma-Aminobutyric Acid glutamate decarboxylase 1 Homo sapiens
2 The pathophysiology of the disease is caused by the decreased level of the glutamic acid decarboxylase (GAD) activity due to an autoantibody against GAD that decreases the level of gamma-aminobutyric acid (GABA). gamma-Aminobutyric Acid glutamate decarboxylase 1 Homo sapiens
3 The pathophysiology of the disease is caused by the decreased level of the glutamic acid decarboxylase (GAD) activity due to an autoantibody against GAD that decreases the level of gamma-aminobutyric acid (GABA). gamma-Aminobutyric Acid glutamate decarboxylase 1 Homo sapiens
4 The pathophysiology of the disease is caused by the decreased level of the glutamic acid decarboxylase (GAD) activity due to an autoantibody against GAD that decreases the level of gamma-aminobutyric acid (GABA). gamma-Aminobutyric Acid glutamate decarboxylase 1 Homo sapiens
5 The pathophysiology of the disease is caused by the decreased level of the glutamic acid decarboxylase (GAD) activity due to an autoantibody against GAD that decreases the level of gamma-aminobutyric acid (GABA). gamma-Aminobutyric Acid glutamate decarboxylase 1 Homo sapiens
6 The pathophysiology of the disease is caused by the decreased level of the glutamic acid decarboxylase (GAD) activity due to an autoantibody against GAD that decreases the level of gamma-aminobutyric acid (GABA). gamma-Aminobutyric Acid glutamate decarboxylase 1 Homo sapiens