Title : Chronic exposure of nanomolar MC-LR caused oxidative stress and inflammatory responses in HepG2 cells.

Pub. Date : 2018 Feb

PMID : 29117589






1 Functional Relationships(s)
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1 Additionally, long-term exposure of MC-LR at low concentration remarkably promoted the expression of NF-kappaB p65, COX-2, iNOS, TNF-alpha, IL-1beta, and IL-6 in the cells, suggesting that long-term MC-LR exposure at low concentration can induce inflammatory reaction to HepG2 cells, which might account for MC-induced human hepatitis. Methylcholanthrene mitochondrially encoded cytochrome c oxidase II Homo sapiens