Title : Pro-inflammatory proteins S100A9 and tumor necrosis factor-α suppress erythropoietin elaboration in myelodysplastic syndromes.

Pub. Date : 2017 Dec

PMID : 28983059






2 Functional Relationships(s)
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1 Moreover, in peripheral blood mononuclear cells from patients with myelodysplastic syndromes, lenalidomide significantly reduced steady-state S100A9 generation (P=0.01) and lipopolysaccharide-induced tumor necrosis factor-alpha elaboration (P=0.002). Lenalidomide tumor necrosis factor Homo sapiens
2 Moreover, baseline serum tumor necrosis factor-alpha concentration was significantly higher in responders to erythroid-stimulating agents (P=0.03), whereas lenalidomide responders had significantly lower tumor necrosis factor-alpha and higher S100A9 serum concentrations (P=0.03). Lenalidomide tumor necrosis factor Homo sapiens