Title : Alzheimer's Disease as the Product of a Progressive Energy Deficiency Syndrome in the Central Nervous System: The Neuroenergetic Hypothesis.

Pub. Date : 2017

PMID : 28946565






1 Functional Relationships(s)
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1 More specifically, the age-related decline in the ability of glucose to cross the blood-brain barrier creates a metabolic stress that shifts the normal, benign processing of amyloid-beta protein precursor toward pathways associated with the production of amyloid-beta plaques and tau-containing neurofibrillary tangles that are characteristic of the disease. Glucose microtubule associated protein tau Homo sapiens