Title : Smoking Cessation as a Possible Risk Factor for the Development of Aspirin-Exacerbated Respiratory Disease in Smokers.

Pub. Date : 2018 Jan - Feb

PMID : 28583479






2 Functional Relationships(s)
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1 BACKGROUND: The pathogenesis of aspirin-exacerbated respiratory disease (AERD) is characterized by the low expression of cyclooxygenase-2 (COX-2) in airway epithelia, which decreases the production of prostaglandin E2 (PGE2). Aspirin prostaglandin-endoperoxide synthase 2 Homo sapiens
2 BACKGROUND: The pathogenesis of aspirin-exacerbated respiratory disease (AERD) is characterized by the low expression of cyclooxygenase-2 (COX-2) in airway epithelia, which decreases the production of prostaglandin E2 (PGE2). Aspirin prostaglandin-endoperoxide synthase 2 Homo sapiens