Title : Endothelial damage effects of circulating microparticles from patients with stable angina are reduced by aspirin through ERK/p38 MAPKs pathways.

Pub. Date : 2017 Aug

PMID : 28520220






1 Functional Relationships(s)
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1 CONCLUSION: These results indicate that the pro-inflammatory, oxidative stress, procoagulant, and adhesion properties of MPs can be partly blocked by aspirin via the ERK-NO/O2- and p38 -NF-kappaB-VCAM-1 signal pathway, which clarified other functions beyond anti-atherothrombotic of aspirin. Aspirin nuclear factor kappa B subunit 1 Homo sapiens