Title : LiCl Treatment Induces Programmed Cell Death of Schwannoma Cells through AKT- and MTOR-Mediated Necroptosis.

Pub. Date : 2017 Aug

PMID : 28397069






4 Functional Relationships(s)
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1 LiCl Treatment Induces Programmed Cell Death of Schwannoma Cells through AKT- and MTOR-Mediated Necroptosis. Lithium Chloride AKT serine/threonine kinase 1 Homo sapiens
2 Moreover, LiCl treatment induces ROS generation and activates the AKT/mTOR pathway, which is reversed by necrostatin-1 treatment. Lithium Chloride AKT serine/threonine kinase 1 Homo sapiens
3 Based on our results, LiCl treatment may induce the programmed cell death of schwannoma cells through AKT- and mTOR-mediated necroptosis, potentially representing a new mechanism by which LiCl induces tumor cell death. Lithium Chloride AKT serine/threonine kinase 1 Homo sapiens
4 Based on our results, LiCl treatment may induce the programmed cell death of schwannoma cells through AKT- and mTOR-mediated necroptosis, potentially representing a new mechanism by which LiCl induces tumor cell death. Lithium Chloride AKT serine/threonine kinase 1 Homo sapiens