Title : Effect of nitric oxide to axonal degeneration in multiple sclerosis via downregulating monocarboxylate transporter 1 in oligodendrocytes.

Pub. Date : 2017 Jul 1

PMID : 28392448






5 Functional Relationships(s)
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Sentence
Compound Name
Protein Name
Organism
1 Inadequate glucose causes monocarboxylate transporter 1 (MCT1) to transfer lactate from oligodendrocytes (OLs) to neurons, which decreases MCT1 and results in energy substrate deficit (mainly lactate) in axons. Lactic Acid solute carrier family 16 member 1 Homo sapiens
2 Inadequate glucose causes monocarboxylate transporter 1 (MCT1) to transfer lactate from oligodendrocytes (OLs) to neurons, which decreases MCT1 and results in energy substrate deficit (mainly lactate) in axons. Lactic Acid solute carrier family 16 member 1 Homo sapiens
3 Inadequate glucose causes monocarboxylate transporter 1 (MCT1) to transfer lactate from oligodendrocytes (OLs) to neurons, which decreases MCT1 and results in energy substrate deficit (mainly lactate) in axons. Lactic Acid solute carrier family 16 member 1 Homo sapiens
4 Inadequate glucose causes monocarboxylate transporter 1 (MCT1) to transfer lactate from oligodendrocytes (OLs) to neurons, which decreases MCT1 and results in energy substrate deficit (mainly lactate) in axons. Lactic Acid solute carrier family 16 member 1 Homo sapiens
5 Inadequate glucose causes monocarboxylate transporter 1 (MCT1) to transfer lactate from oligodendrocytes (OLs) to neurons, which decreases MCT1 and results in energy substrate deficit (mainly lactate) in axons. Lactic Acid solute carrier family 16 member 1 Homo sapiens