Title : 17β-estradiol-induced ACSL4 protein expression promotes an invasive phenotype in estrogen receptor positive mammary carcinoma cells.

Pub. Date : 2017 Apr 1

PMID : 28334272






1 Functional Relationships(s)
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1 In this study, treatment of steroid-starved ERalpha-positive MCF-7 and T47D mammary carcinoma cells with 17beta-estradiol resulted in increased cellular uptake of the PUFA arachidonic acid (AA) and eicosapentaenoic acid (EPA), important building blocks for cellular membranes, and increased ACSL4 protein levels. Steroids estrogen receptor 1 Homo sapiens