Title : Pinocembrin Suppresses H2O2-Induced Mitochondrial Dysfunction by a Mechanism Dependent on the Nrf2/HO-1 Axis in SH-SY5Y Cells.

Pub. Date : 2018 Feb

PMID : 28084593






2 Functional Relationships(s)
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1 Furthermore, PB induced anti-inflammatory effects by abolishing the H2O2-dependent activation of the nuclear factor-kappaB (NF-kappaB) and upregulation of interleukin-1beta (IL-1beta) and tumor necrosis factor-alpha (TNF-alpha). pinocembrin interleukin 1 beta Homo sapiens
2 Furthermore, PB induced anti-inflammatory effects by abolishing the H2O2-dependent activation of the nuclear factor-kappaB (NF-kappaB) and upregulation of interleukin-1beta (IL-1beta) and tumor necrosis factor-alpha (TNF-alpha). pinocembrin interleukin 1 beta Homo sapiens