Title : Mitochondrial involvement in skeletal muscle insulin resistance: A case of imbalanced bioenergetics.

Pub. Date : 2016 Oct

PMID : 27473535






2 Functional Relationships(s)
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1 This market-economic control of myocellular bioenergetics offers a mechanism by which insulin-signalling deficiency can cause apparent mitochondrial dysfunction, as insulin resistance lowers skeletal muscle anabolism and thus dampens ATP demand and, consequently, oxidative ATP synthesis. Adenosine Triphosphate insulin Homo sapiens
2 This market-economic control of myocellular bioenergetics offers a mechanism by which insulin-signalling deficiency can cause apparent mitochondrial dysfunction, as insulin resistance lowers skeletal muscle anabolism and thus dampens ATP demand and, consequently, oxidative ATP synthesis. Adenosine Triphosphate insulin Homo sapiens