Title : PML nuclear body disruption impairs DNA double-strand break sensing and repair in APL.

Pub. Date : 2016 Jul 28

PMID : 27468685






4 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 All-trans-retinoic acid (RA) treatment induces PML-RARalpha degradation, restores PML-NB functions, and causes terminal cell differentiation of APL blasts. Tretinoin PML nuclear body scaffold Homo sapiens
2 All-trans-retinoic acid (RA) treatment induces PML-RARalpha degradation, restores PML-NB functions, and causes terminal cell differentiation of APL blasts. Tretinoin PML nuclear body scaffold Homo sapiens
3 All-trans-retinoic acid (RA) treatment induces PML-RARalpha degradation, restores PML-NB functions, and causes terminal cell differentiation of APL blasts. Tretinoin PML nuclear body scaffold Homo sapiens
4 All-trans-retinoic acid (RA) treatment induces PML-RARalpha degradation, restores PML-NB functions, and causes terminal cell differentiation of APL blasts. Tretinoin PML nuclear body scaffold Homo sapiens