Title : PTEN enhances G2/M arrest in etoposide-treated MCF‑7 cells through activation of the ATM pathway.

Pub. Date : 2016 May

PMID : 26986476






1 Functional Relationships(s)
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1 The results showed that knockdown of PTEN strongly antagonized ATM activation in response to etoposide treatment, and thereby reduced the phosphorylation level of ATM substrates, including H2AX, P53 and Chk2. Etoposide tumor protein p53 Homo sapiens