Title : Mechanisms underlying spontaneous constrictions of postcapillary venules in the rat stomach.

Pub. Date : 2016 Feb

PMID : 26530829






3 Functional Relationships(s)
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1 Stellate-shaped PCV mural cells expressing alpha-smooth muscle actin exhibited synchronised spontaneous Ca(2+) transients to develop vasomotion which was abolished by nifedipine (1 muM), cyclopiazonic acid (10 muM), or Ca(2+)-activated Cl(-) channel inhibitors (100 muM niflumic acid, 1 muM T16Ainh-A01). Nifedipine latexin Homo sapiens
2 Stellate-shaped PCV mural cells expressing alpha-smooth muscle actin exhibited synchronised spontaneous Ca(2+) transients to develop vasomotion which was abolished by nifedipine (1 muM), cyclopiazonic acid (10 muM), or Ca(2+)-activated Cl(-) channel inhibitors (100 muM niflumic acid, 1 muM T16Ainh-A01). Nifedipine latexin Homo sapiens
3 Stellate-shaped PCV mural cells expressing alpha-smooth muscle actin exhibited synchronised spontaneous Ca(2+) transients to develop vasomotion which was abolished by nifedipine (1 muM), cyclopiazonic acid (10 muM), or Ca(2+)-activated Cl(-) channel inhibitors (100 muM niflumic acid, 1 muM T16Ainh-A01). Nifedipine latexin Homo sapiens