Title : Alarmin' Immunologists: IL-33 as a Putative Target for Modulating T Cell-Dependent Responses.

Pub. Date : 2015

PMID : 26082774






1 Functional Relationships(s)
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1 At the beginning, it was known as one of the participants during the development of allergic states and other Th2-mediated responses and it is now accepted that IL-33 contributes to Th1-driven pathologies as demonstrated in animal models of experimental autoimmune encephalomyelitis (EAE), collagen-induced arthritis, and trinitrobenzene sulfonic acid-induced experimental colitis, among others. Trinitrobenzenesulfonic Acid interleukin 33 Homo sapiens