Title : Effect of lithium chloride on endoplasmic reticulum stress-related PERK/ROCK signaling in a rat model of glaucoma.

Pub. Date : 2014 Dec

PMID : 25951661






4 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 Lithium chloride (LiCl) inhibits glycogen synthase kinase-3 beta (GSK-3beta) and attends PERK-induced endoplasmic reticulum stress (ERs) transition. Lithium Chloride glycogen synthase kinase 3 beta Rattus norvegicus
2 Lithium chloride (LiCl) inhibits glycogen synthase kinase-3 beta (GSK-3beta) and attends PERK-induced endoplasmic reticulum stress (ERs) transition. Lithium Chloride glycogen synthase kinase 3 beta Rattus norvegicus
3 Lithium chloride (LiCl) inhibits glycogen synthase kinase-3 beta (GSK-3beta) and attends PERK-induced endoplasmic reticulum stress (ERs) transition. Lithium Chloride glycogen synthase kinase 3 beta Rattus norvegicus
4 Lithium chloride (LiCl) inhibits glycogen synthase kinase-3 beta (GSK-3beta) and attends PERK-induced endoplasmic reticulum stress (ERs) transition. Lithium Chloride glycogen synthase kinase 3 beta Rattus norvegicus