Title : Endothelial function, folate pharmacogenomics, and neurocognition in psychotic disorders.

Pub. Date : 2015 May

PMID : 25728832






2 Functional Relationships(s)
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1 The current study examined folate metabolism as a potential mechanism of CVD and neurocognitive deficits by: 1) using endothelial dysfunction as a biomarker of CVD, and 2) comparing enzymes associated with neurocognition, CVD, and critical to folate metabolism, methylenetetrahydrofolate reductase (MTHFR) and catechol-o-methyl transferase (COMT). Folic Acid catechol-O-methyltransferase Homo sapiens
2 The current study examined folate metabolism as a potential mechanism of CVD and neurocognitive deficits by: 1) using endothelial dysfunction as a biomarker of CVD, and 2) comparing enzymes associated with neurocognition, CVD, and critical to folate metabolism, methylenetetrahydrofolate reductase (MTHFR) and catechol-o-methyl transferase (COMT). Folic Acid catechol-O-methyltransferase Homo sapiens