Title : Tumor necrosis factor as an activator of human granulocytes. Potentiation of the metabolisms triggered by the Ca2+-mobilizing agonists.

Pub. Date : 1989 Mar 1

PMID : 2537361






1 Functional Relationships(s)
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1 TNF-induced O2- release was inhibited by cAMP agonists or ionomycin, and was not accompanied with an increase in cytoplasmic free Ca2+ [( Ca2+]i) and membrane potential changes (depolarization). Ionomycin tumor necrosis factor Homo sapiens