Title : Retinoic acid controls body axis extension by directly repressing Fgf8 transcription.

Pub. Date : 2014 Aug

PMID : 25053430






1 Functional Relationships(s)
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Protein Name
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1 The co-regulator RERE, the loss of which results in ectopic Fgf8 expression and somite defects, was recruited near the RARb RARE by RA, but was released from the Fgf8 RARE by RA. Tretinoin retinoic acid receptor, beta Mus musculus