Pub. Date : 2014 Nov
PMID : 25014355
3 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
1 | Testosterone treatment caused up-regulation of proapoptotic genes Bax (Bcl-2-associated X protein), p53 (tumor protein p53), and JNK (c-Jun N-terminal kinases) in both cell lines and increased expression of Bax, Caspase 3, and cytochrome C proteins. | Testosterone | mitogen-activated protein kinase 8 | Homo sapiens |
2 | Testosterone treatment caused up-regulation of proapoptotic genes Bax (Bcl-2-associated X protein), p53 (tumor protein p53), and JNK (c-Jun N-terminal kinases) in both cell lines and increased expression of Bax, Caspase 3, and cytochrome C proteins. | Testosterone | mitogen-activated protein kinase 8 | Homo sapiens |
3 | Treatment with a zinc chelator or a MAPK inhibitor blocked testosterone-induced increases in Bax, p53, and JNK mRNA expression. | Testosterone | mitogen-activated protein kinase 8 | Homo sapiens |