Title : Transgenic mice overexpressing amyloid precursor protein exhibit early metabolic deficits and a pathologically low leptin state associated with hypothalamic dysfunction in arcuate neuropeptide Y neurons.

Pub. Date : 2014 Jul 2

PMID : 24990930






1 Functional Relationships(s)
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1 In addition, arcuate NPY neurons exhibited abnormal electrophysiological responses to leptin in Tg2576 hypothalamic slices or wild-type slices treated with Abeta. tg2576 leptin Mus musculus