Title : Selective activation of hTRPV1 by N-geranyl cyclopropylcarboxamide, an amiloride-insensitive salt taste enhancer.

Pub. Date : 2014

PMID : 24586504






4 Functional Relationships(s)
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1 NGCC-induced Ca(2+) influx was significantly attenuated by ruthenium red (RR; 30 microM), a non-specific blocker of TRP channels and capsazepine (CZP; 5 microM), a specific antagonist of TRPV1, implying that NGCC directly activates hTRPV1. Ruthenium Red transient receptor potential cation channel subfamily V member 1 Homo sapiens
2 NGCC-induced Ca(2+) influx was significantly attenuated by ruthenium red (RR; 30 microM), a non-specific blocker of TRP channels and capsazepine (CZP; 5 microM), a specific antagonist of TRPV1, implying that NGCC directly activates hTRPV1. Ruthenium Red transient receptor potential cation channel subfamily V member 1 Homo sapiens
3 NGCC-induced Ca(2+) influx was significantly attenuated by ruthenium red (RR; 30 microM), a non-specific blocker of TRP channels and capsazepine (CZP; 5 microM), a specific antagonist of TRPV1, implying that NGCC directly activates hTRPV1. Ruthenium Red transient receptor potential cation channel subfamily V member 1 Homo sapiens
4 NGCC-induced Ca(2+) influx was significantly attenuated by ruthenium red (RR; 30 microM), a non-specific blocker of TRP channels and capsazepine (CZP; 5 microM), a specific antagonist of TRPV1, implying that NGCC directly activates hTRPV1. Ruthenium Red transient receptor potential cation channel subfamily V member 1 Homo sapiens