Title : Overexpression of atypical protein kinase C in HeLa cells facilitates macropinocytosis via Src activation.

Pub. Date : 2014 Jun

PMID : 24582589






2 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 Src-aPKC interaction was essential; substitution of the proline residues in aPKC that associate with the Src-SH3 binding domain rendered the mutant kinase unable to induce macropinocytosis in transfected cells. Proline SRC proto-oncogene, non-receptor tyrosine kinase Homo sapiens
2 Src-aPKC interaction was essential; substitution of the proline residues in aPKC that associate with the Src-SH3 binding domain rendered the mutant kinase unable to induce macropinocytosis in transfected cells. Proline SRC proto-oncogene, non-receptor tyrosine kinase Homo sapiens