Pub. Date : 2014 Feb 5
PMID : 24501372
6 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
| 1 | Abnormal intracellular calcium signaling and SNARE-dependent exocytosis contributes to SOD1G93A astrocyte-mediated toxicity in amyotrophic lateral sclerosis. | Calcium | superoxide dismutase 1 | Homo sapiens |
| 2 | We found that purinergic stimulation induces excess calcium release from the ER stores in SOD1G93A astrocytes, which results from the abnormal ER calcium accumulation and is independent of clearance mechanisms. | Calcium | superoxide dismutase 1 | Homo sapiens |
| 3 | We found that purinergic stimulation induces excess calcium release from the ER stores in SOD1G93A astrocytes, which results from the abnormal ER calcium accumulation and is independent of clearance mechanisms. | Calcium | superoxide dismutase 1 | Homo sapiens |
| 4 | We found that oxidant-induced increased S-glutathionylation and calcium-independent puncta formation of the ER calcium sensor STIM1 underlies the abnormal SOCE response in SOD1G93A astrocytes. | Calcium | superoxide dismutase 1 | Homo sapiens |
| 5 | We found that oxidant-induced increased S-glutathionylation and calcium-independent puncta formation of the ER calcium sensor STIM1 underlies the abnormal SOCE response in SOD1G93A astrocytes. | Calcium | superoxide dismutase 1 | Homo sapiens |
| 6 | Enhanced SOCE contributes to ER calcium overload in SOD1G93A astrocytes and excess calcium release from the ER during ATP stimulation. | Calcium | superoxide dismutase 1 | Homo sapiens |