Pub. Date : 2013 Dec
PMID : 24190484
6 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
| 1 | Remifentanil preconditioning alleviating brain damage of cerebral ischemia reperfusion rats by regulating the JNK signal pathway and TNF-alpha/TNFR1 signal pathway. | Remifentanil | tumor necrosis factor | Rattus norvegicus |
| 2 | TNF-alpha and TNFR1 in CIR + Remifentanil group rats was found to be significant lower than that in CIR group rats. | Remifentanil | tumor necrosis factor | Rattus norvegicus |
| 3 | The inhibitory effects of Remifentanil preconditioning on the brain damage are achieved probably through blocking the activation of TNF-alpha/TNFR1, JNK signal transduction pathways, which implies that Remifentanil preconditioning may be a potential and effective way for prevention of the ischemic/reperfusion injury through the suppression extrinsic apoptotic signal pathway induced by TNF-alpha/TNFR1, JNK signal pathways. | Remifentanil | tumor necrosis factor | Rattus norvegicus |
| 4 | The inhibitory effects of Remifentanil preconditioning on the brain damage are achieved probably through blocking the activation of TNF-alpha/TNFR1, JNK signal transduction pathways, which implies that Remifentanil preconditioning may be a potential and effective way for prevention of the ischemic/reperfusion injury through the suppression extrinsic apoptotic signal pathway induced by TNF-alpha/TNFR1, JNK signal pathways. | Remifentanil | tumor necrosis factor | Rattus norvegicus |
| 5 | The inhibitory effects of Remifentanil preconditioning on the brain damage are achieved probably through blocking the activation of TNF-alpha/TNFR1, JNK signal transduction pathways, which implies that Remifentanil preconditioning may be a potential and effective way for prevention of the ischemic/reperfusion injury through the suppression extrinsic apoptotic signal pathway induced by TNF-alpha/TNFR1, JNK signal pathways. | Remifentanil | tumor necrosis factor | Rattus norvegicus |
| 6 | The inhibitory effects of Remifentanil preconditioning on the brain damage are achieved probably through blocking the activation of TNF-alpha/TNFR1, JNK signal transduction pathways, which implies that Remifentanil preconditioning may be a potential and effective way for prevention of the ischemic/reperfusion injury through the suppression extrinsic apoptotic signal pathway induced by TNF-alpha/TNFR1, JNK signal pathways. | Remifentanil | tumor necrosis factor | Rattus norvegicus |