Title : Mitochondrial impairment increases FL-PINK1 levels by calcium-dependent gene expression.

Pub. Date : 2014 Feb

PMID : 24184327






3 Functional Relationships(s)
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1 In this study, we report that increased mitochondrial PINK1 levels observed in human neuroblastoma SH-SY5Y cells after carbonyl cyanide m-chlorophelyhydrazone (CCCP) treatment were due to de novo protein synthesis, and not just increased stabilization of full length PINK1 (FL-PINK1). Carbonyl Cyanide m-Chlorophenyl Hydrazone PTEN induced kinase 1 Homo sapiens
2 In this study, we report that increased mitochondrial PINK1 levels observed in human neuroblastoma SH-SY5Y cells after carbonyl cyanide m-chlorophelyhydrazone (CCCP) treatment were due to de novo protein synthesis, and not just increased stabilization of full length PINK1 (FL-PINK1). Carbonyl Cyanide m-Chlorophenyl Hydrazone PTEN induced kinase 1 Homo sapiens
3 In this study, we report that increased mitochondrial PINK1 levels observed in human neuroblastoma SH-SY5Y cells after carbonyl cyanide m-chlorophelyhydrazone (CCCP) treatment were due to de novo protein synthesis, and not just increased stabilization of full length PINK1 (FL-PINK1). Carbonyl Cyanide m-Chlorophenyl Hydrazone PTEN induced kinase 1 Homo sapiens