Title : Induction of mitochondrial permeability transition (MPT) pore opening and ROS formation as a mechanism for methamphetamine-induced mitochondrial toxicity.

Pub. Date : 2014 Jan

PMID : 24062016






1 Functional Relationships(s)
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1 In addition, collapse of mitochondrial membrane potential, mitochondrial swelling, and release of cytochrome c following METH treatment were well inhibited by pretreatment of mitochondria with cyclosporin A and butylated hydroxytoluene. Butylated Hydroxytoluene cytochrome c, somatic Homo sapiens