Title : Monoamine oxidases are mediators of endothelial dysfunction in the mouse aorta.

Pub. Date : 2013 Jul

PMID : 23670301






1 Functional Relationships(s)
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1 Histamine stimulation of human umbilical vein endothelial cells to activate endothelial NO synthase resulted in an increase in cyclic GMP, which was almost abrogated by MAO-A exposure. Histamine monoamine oxidase A Homo sapiens