Title : Study of the cellular mechanism of Sunitinib mediated inactivation of activated hepatic stellate cells and its implications in angiogenesis.

Pub. Date : 2013 Apr 5

PMID : 23454556






1 Functional Relationships(s)
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1 In conclusion, the present study demonstrates that the RTK inhibitor Sunitinib blocks the activation of HSC and angiogenesis suggesting its potential as a drug candidate in pathological conditions like liver fibrosis and hepatocellular carcinoma. Sunitinib ret proto-oncogene Homo sapiens