Title : Interleukin-1 receptor and caspase-1 are required for the Th17 response in nitrogen dioxide-promoted allergic airway disease.

Pub. Date : 2013 May

PMID : 23371061






2 Functional Relationships(s)
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1 NO2 exposure is capable of allergically sensitizing mice to the innocuous inhaled antigen ovalbumin (OVA), promoting neutrophil and eosinophil recruitment, and a mixed Th2/Th17 response upon antigen challenge that is reminiscent of severe asthma. Nitrogen Dioxide serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus
2 NO2 exposure is capable of allergically sensitizing mice to the innocuous inhaled antigen ovalbumin (OVA), promoting neutrophil and eosinophil recruitment, and a mixed Th2/Th17 response upon antigen challenge that is reminiscent of severe asthma. Nitrogen Dioxide serine (or cysteine) peptidase inhibitor, clade B, member 1, pseudogene Mus musculus