Title : Adrenergic modulation of potassium metabolism in uremia.

Pub. Date : 1990 Feb

PMID : 2308263






3 Functional Relationships(s)
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1 Thus, the higher rise in potassium concentration during exercise with propranolol could only be explained by adrenergic blockade at the beta-2 receptor site. Potassium potassium calcium-activated channel subfamily M regulatory beta subunit 2 Homo sapiens
2 These results support the concept that adrenergic control of extrarenal potassium homeostasis in dialysis patients is mediated at the beta-2 receptor. Potassium potassium calcium-activated channel subfamily M regulatory beta subunit 2 Homo sapiens
3 Since a deterioration in potassium homeostasis during exercise is observed with beta-2, but not beta-1 blockade, selective beta-1 adrenergic blocking agents may be safer in dialysis patients. Potassium potassium calcium-activated channel subfamily M regulatory beta subunit 2 Homo sapiens