Title : The role of fatty acid amide hydrolase inhibition in nicotine reward and dependence.

Pub. Date : 2013 Mar 19

PMID : 22705310






4 Functional Relationships(s)
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Protein Name
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1 FAAH KO mice and animals treated with FAAH inhibitors are impaired in their ability to hydrolyze AEA and other non-cannabinoid lipid signaling molecules, such as oleoylethanolamide (OEA) and palmitoylethanolamide (PEA). oleoylethanolamide fatty acid amide hydrolase Mus musculus
2 FAAH KO mice and animals treated with FAAH inhibitors are impaired in their ability to hydrolyze AEA and other non-cannabinoid lipid signaling molecules, such as oleoylethanolamide (OEA) and palmitoylethanolamide (PEA). oleoylethanolamide fatty acid amide hydrolase Mus musculus
3 FAAH KO mice and animals treated with FAAH inhibitors are impaired in their ability to hydrolyze AEA and other non-cannabinoid lipid signaling molecules, such as oleoylethanolamide (OEA) and palmitoylethanolamide (PEA). oleoylethanolamide fatty acid amide hydrolase Mus musculus
4 FAAH KO mice and animals treated with FAAH inhibitors are impaired in their ability to hydrolyze AEA and other non-cannabinoid lipid signaling molecules, such as oleoylethanolamide (OEA) and palmitoylethanolamide (PEA). oleoylethanolamide fatty acid amide hydrolase Mus musculus