Title : Mechanisms shaping the slow nicotinic synaptic current at the motoneuron-renshaw cell synapse.

Pub. Date : 2012 Jun 13

PMID : 22699921






1 Functional Relationships(s)
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1 The pharmacological inactivation of AChE by neostigmine caused the appearance of an ultra-slow (second range) decay component in eEPSCs and also a continuous inward current interpreted as resulting from a continuous ACh presence. Neostigmine acetylcholinesterase Mus musculus