Title : Increasing levels of wild-type CREB up-regulates several activity-regulated inhibitor of death (AID) genes and promotes neuronal survival.

Pub. Date : 2012 May 18

PMID : 22607375






3 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 The observed degradation of CREB protein following NMDA treatment of hippocampal neurons suggests that the known CREB shut-off associated with extrasynaptic NMDA receptor-induced excitotoxicity is followed by CREB proteolysis. N-Methylaspartate cAMP responsive element binding protein 1 Mus musculus
2 The observed degradation of CREB protein following NMDA treatment of hippocampal neurons suggests that the known CREB shut-off associated with extrasynaptic NMDA receptor-induced excitotoxicity is followed by CREB proteolysis. N-Methylaspartate cAMP responsive element binding protein 1 Mus musculus
3 The observed degradation of CREB protein following NMDA treatment of hippocampal neurons suggests that the known CREB shut-off associated with extrasynaptic NMDA receptor-induced excitotoxicity is followed by CREB proteolysis. N-Methylaspartate cAMP responsive element binding protein 1 Mus musculus