Title : High-sugar intake does not exacerbate metabolic abnormalities or cardiac dysfunction in genetic cardiomyopathy.

Pub. Date : 2012 May

PMID : 22304857






5 Functional Relationships(s)
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Sentence
Compound Name
Protein Name
Organism
1 Glucose-6-phosphate dehydrogenase (G6PD) can fuel ROS production by providing reduced nicotinamide adenine dinucleotide phosphate (NADPH) for superoxide generation by NADPH oxidase. NADP glucose-6-phosphate dehydrogenase Homo sapiens
2 Glucose-6-phosphate dehydrogenase (G6PD) can fuel ROS production by providing reduced nicotinamide adenine dinucleotide phosphate (NADPH) for superoxide generation by NADPH oxidase. NADP glucose-6-phosphate dehydrogenase Homo sapiens
3 Glucose-6-phosphate dehydrogenase (G6PD) can fuel ROS production by providing reduced nicotinamide adenine dinucleotide phosphate (NADPH) for superoxide generation by NADPH oxidase. NADP glucose-6-phosphate dehydrogenase Homo sapiens
4 Glucose-6-phosphate dehydrogenase (G6PD) can fuel ROS production by providing reduced nicotinamide adenine dinucleotide phosphate (NADPH) for superoxide generation by NADPH oxidase. NADP glucose-6-phosphate dehydrogenase Homo sapiens
5 We hypothesized that a high-sugar intake would increase flux through G6PD to increase myocardial NADPH and ROS and accelerate cardiac dysfunction and death. NADP glucose-6-phosphate dehydrogenase Homo sapiens