Title : Urinary prostaglandin E2 metabolite and risk for colorectal adenoma.

Pub. Date : 2012 Feb

PMID : 22166248






4 Functional Relationships(s)
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1 Most of the COX-2 tumor-inducing effects are believed to be mediated through overproduction of prostaglandin E(2) (PGE(2)), which can be measured using a urinary metabolite of PGE(2), PGE-M. Urinary PGE-M was assessed in a case-control study of colorectal adenoma. Prostaglandins E mitochondrially encoded cytochrome c oxidase II Homo sapiens
2 Most of the COX-2 tumor-inducing effects are believed to be mediated through overproduction of prostaglandin E(2) (PGE(2)), which can be measured using a urinary metabolite of PGE(2), PGE-M. Urinary PGE-M was assessed in a case-control study of colorectal adenoma. Prostaglandins E mitochondrially encoded cytochrome c oxidase II Homo sapiens
3 Most of the COX-2 tumor-inducing effects are believed to be mediated through overproduction of prostaglandin E(2) (PGE(2)), which can be measured using a urinary metabolite of PGE(2), PGE-M. Urinary PGE-M was assessed in a case-control study of colorectal adenoma. Prostaglandins E mitochondrially encoded cytochrome c oxidase II Homo sapiens
4 Most of the COX-2 tumor-inducing effects are believed to be mediated through overproduction of prostaglandin E(2) (PGE(2)), which can be measured using a urinary metabolite of PGE(2), PGE-M. Urinary PGE-M was assessed in a case-control study of colorectal adenoma. Prostaglandins E mitochondrially encoded cytochrome c oxidase II Homo sapiens