Title : Rosiglitazone reduces fatty acid translocase and increases AMPK in skeletal muscle in aged rats: a possible mechanism to prevent high-fat-induced insulin resistance.

Pub. Date : 2010 Sep

PMID : 21034554






4 Functional Relationships(s)
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1 BACKGROUND: As an agonist of peroxisome proliferator-activated receptor-gamma (PPARgamma), rosiglitazone can prevent acute fatty acid-induced insulin resistance in rats, however, the precise mechanisms by which rosiglitazone alleviates insulin resistance induced by high-fat diet need to be further investigated. Rosiglitazone peroxisome proliferator-activated receptor gamma Rattus norvegicus
2 BACKGROUND: As an agonist of peroxisome proliferator-activated receptor-gamma (PPARgamma), rosiglitazone can prevent acute fatty acid-induced insulin resistance in rats, however, the precise mechanisms by which rosiglitazone alleviates insulin resistance induced by high-fat diet need to be further investigated. Rosiglitazone peroxisome proliferator-activated receptor gamma Rattus norvegicus
3 BACKGROUND: As an agonist of peroxisome proliferator-activated receptor-gamma (PPARgamma), rosiglitazone can prevent acute fatty acid-induced insulin resistance in rats, however, the precise mechanisms by which rosiglitazone alleviates insulin resistance induced by high-fat diet need to be further investigated. Rosiglitazone peroxisome proliferator-activated receptor gamma Rattus norvegicus
4 BACKGROUND: As an agonist of peroxisome proliferator-activated receptor-gamma (PPARgamma), rosiglitazone can prevent acute fatty acid-induced insulin resistance in rats, however, the precise mechanisms by which rosiglitazone alleviates insulin resistance induced by high-fat diet need to be further investigated. Rosiglitazone peroxisome proliferator-activated receptor gamma Rattus norvegicus