Title : Hyperpolarization-activated currents in gonadotropin-releasing hormone (GnRH) neurons contribute to intrinsic excitability and are regulated by gonadal steroid feedback.

Pub. Date : 2010 Oct 6

PMID : 20926664






6 Functional Relationships(s)
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1 Hyperpolarization-activated currents in gonadotropin-releasing hormone (GnRH) neurons contribute to intrinsic excitability and are regulated by gonadal steroid feedback. Steroids gonadotropin releasing hormone 1 Mus musculus
2 Hyperpolarization-activated currents in gonadotropin-releasing hormone (GnRH) neurons contribute to intrinsic excitability and are regulated by gonadal steroid feedback. Steroids gonadotropin releasing hormone 1 Mus musculus
3 Pulsatile release of gonadotropin-releasing hormone (GnRH) is required for fertility and is regulated by steroid feedback. Steroids gonadotropin releasing hormone 1 Mus musculus
4 Pulsatile release of gonadotropin-releasing hormone (GnRH) is required for fertility and is regulated by steroid feedback. Steroids gonadotropin releasing hormone 1 Mus musculus
5 As estradiol is the primary central negative feedback hormone on GnRH neuron firing in males, this provides insight into the mechanisms by which steroid hormones potentially alter the intrinsic properties of GnRH neurons to change their activity. Steroids gonadotropin releasing hormone 1 Mus musculus
6 As estradiol is the primary central negative feedback hormone on GnRH neuron firing in males, this provides insight into the mechanisms by which steroid hormones potentially alter the intrinsic properties of GnRH neurons to change their activity. Steroids gonadotropin releasing hormone 1 Mus musculus