Title : XPC silencing sensitizes glioma cells to arsenic trioxide via increased oxidative damage.

Pub. Date : 2010 Jul

PMID : 20403967






5 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 XPC silencing sensitizes glioma cells to arsenic trioxide via increased oxidative damage. Arsenic Trioxide XPC complex subunit, DNA damage recognition and repair factor Homo sapiens
2 We found that arsenic trioxide (ATO), like ultraviolet (UV) irradiation, induced the expression of xeroderma pigmentosum group C (XPC) but not of xeroderma pigmentosum A in a human glioma cell line, U87. Arsenic Trioxide XPC complex subunit, DNA damage recognition and repair factor Homo sapiens
3 We found that arsenic trioxide (ATO), like ultraviolet (UV) irradiation, induced the expression of xeroderma pigmentosum group C (XPC) but not of xeroderma pigmentosum A in a human glioma cell line, U87. Arsenic Trioxide XPC complex subunit, DNA damage recognition and repair factor Homo sapiens
4 We found that arsenic trioxide (ATO), like ultraviolet (UV) irradiation, induced the expression of xeroderma pigmentosum group C (XPC) but not of xeroderma pigmentosum A in a human glioma cell line, U87. Arsenic Trioxide XPC complex subunit, DNA damage recognition and repair factor Homo sapiens
5 We found that arsenic trioxide (ATO), like ultraviolet (UV) irradiation, induced the expression of xeroderma pigmentosum group C (XPC) but not of xeroderma pigmentosum A in a human glioma cell line, U87. Arsenic Trioxide XPC complex subunit, DNA damage recognition and repair factor Homo sapiens