Title : Upregulation of Nox4 by hypertrophic stimuli promotes apoptosis and mitochondrial dysfunction in cardiac myocytes.

Pub. Date : 2010 Apr 16

PMID : 20185797






2 Functional Relationships(s)
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Sentence
Compound Name
Protein Name
Organism
1 Nox4 is primarily localized in mitochondria and upregulation of Nox4 enhanced both rotenone- and diphenyleneiodonium-sensitive O(2)(-) production in mitochondria. diphenyleneiodonium NADPH oxidase 4 Mus musculus
2 Nox4 is primarily localized in mitochondria and upregulation of Nox4 enhanced both rotenone- and diphenyleneiodonium-sensitive O(2)(-) production in mitochondria. diphenyleneiodonium NADPH oxidase 4 Mus musculus